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Immunohistochemical study for the expression of leukocyte adhesion molecules, and FGF23 and ACE2 in P. gingivalis LPS-induced diabetic nephropathy | BMC Nephrology | Full Text
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LPS from Porphyromonas gingivalis increases the sensitivity of contractile response mediated by endothelin-B (ETB) receptors in cultured endothelium-intact rat coronary arteries - ScienceDirect
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Porphyromonas gingivalis lipopolysaccharide increases lipid accumulation by affecting CD36 and ATP-binding cassette transporter A1 in macrophages
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Porphyromonas gingivalis lipopolysaccharides act exclusively through TLR4 with a resilience between mouse and human | Scientific Reports
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Structures of previously characterized P. gingivalis lipid A mass ions.... | Download Scientific Diagram
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Mechanism of <i>Porphyromonas gingivalis</i>-lipopolysaccharide in regulating the insulin signaling pathway in adipocytes via X-box binding protein 1
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Identification of an O‐antigen chain length regulator, WzzP, in Porphyromonas gingivalis - Shoji - 2013 - MicrobiologyOpen - Wiley Online Library
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Porphyromonas gingivalis induces penetration of lipopolysaccharide and peptidoglycan through the gingival epithelium via degradation of coxsackievirus and adenovirus receptor - Takeuchi - 2021 - Cellular Microbiology - Wiley Online Library
Innate immunomodulation by lipophilic termini of lipopolysaccharide; synthesis of lipid As from Porphyromonas gingivalis and other bacteria and their immunomodulative responses - Molecular BioSystems (RSC Publishing)
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Author Correction: Heterogeneous Porphyromonas gingivalis LPS modulates immuno-inflammatory response, antioxidant defense and cytoskeletal dynamics in human gingival fibroblasts | Scientific Reports
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Changes in lipopolysaccharide profile of Porphyromonas gingivalis clinical isolates correlate with changes in colony morphology and polymyxin B resistance - ScienceDirect
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Free Lipid A Isolated from Porphyromonas gingivalis Lipopolysaccharide Is Contaminated with Phosphorylated Dihydroceramide Lipids: Recovery in Diseased Dental Samples | Infection and Immunity
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Porphyromonas gingivalis lipopolysaccharide induces cognitive dysfunction, mediated by neuronal inflammation via activation of the TLR4 signaling pathway in C57BL/6 mice | Journal of Neuroinflammation | Full Text
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Immunological Pathways Triggered by <i>Porphyromonas gingivalis</i> and <i>Fusobacterium nucleatum</i>: Therapeutic Possibilities? - Abstract - Europe PMC
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Porphyromonas gingivalis lipopolysaccharide promotes T-hel per17 cell differentiation by upregulating Delta-like ligand 4 expression on CD14+ monocytes [PeerJ]
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Schematic structure of lipopolysaccharide (LPS) of the outer membrane... | Download Scientific Diagram
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Frontiers | Pathogenesis of Important Virulence Factors of Porphyromonas gingivalis via Toll-Like Receptors | Cellular and Infection Microbiology
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Porphyromonas gingivalis lipopolysaccharides act exclusively through TLR4 with a resilience between mouse and human | Scientific Reports
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Porphyromonas gingivalis Stimulates Bone Resorption by Enhancing RANKL (Receptor Activator of NF-κB Ligand) through Activation of Toll-like Receptor 2 in Osteoblasts* - Journal of Biological Chemistry
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Synthetic tetra-acylated derivatives of lipid A from Porphyromonas gingivalis are antagonists of human TLR4 - Organic & Biomolecular Chemistry (RSC Publishing) DOI:10.1039/B809090D
Porphyromonas gingivalis induces penetration of lipopolysaccharide and peptidoglycan through the gingival epithelium via degradation of junctional adhesion molecule 1 | PLOS Pathogens
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Pathogens | Free Full-Text | Porphyromonas gingivalis and Its Systemic Impact: Current Status | HTML
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